Gastric cancer

Francis Peyton Rous' Co-carcinogenesis hypothesis: that a virus and a carcinogen together cause cancer. (1966 Nobel prize for HPV work)  http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2135410/

My co-carcinogenesis takes his further because carcinogens inhibit polymerases.

What I surmise from his hypothesis:

Cancer occurs because a carcinogen inhibits the viral polymerase better than the human polymerase. When a virus infects the same cell as a carcinogen they interact.  Instead of the virus making what it wants in the host the infected cell is transformed into a cancer cell by the carcinogen. 

For gastric cancer the carcinogen is created  by H.pylori in most cases.

Carcinogen from H.pylori and stomach cancer
https://www.cancer.gov/about-cancer/causes-prevention/risk/infectious-agents/h-pylori-fact-sheet

EBV and gastric cancer
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3528272/


Hypothesis:
EBV causes methylation which awakens the embryo genes: herv H

Herv H and gastric cancer
http://onlinelibrary.wiley.com/store/10.1002/ijc.22826/asset/22826_ftp.pdf?v=1&t=j0edcq1b&s=de3efc844253ae648487cb4622d77c57688b1b4b

adenocarcinomas and herv H
https://www.ncbi.nlm.nih.gov/pubmed/26260344


Retroviruses like HIV can also cause a gastric cancer called Gastrointestinal stromal tumor

HIV and gastric cancer
http://www.medscape.com/viewarticle/740694

Herv k and gastric cancer
https://insights.ovid.com/gastroenterology-hepatology/jgahe/2012/12/005/methylation-level-herv-associated-gastric-cancer/1303/00001753

Hypomethylation and gastric cancer
https://www.ncbi.nlm.nih.gov/pubmed/27139434
https://www.ncbi.nlm.nih.gov/pubmed/20978145

prediction: the Herv H EBV gastric cancer is slow growing while the Herv K with HIV will be fast