Is lichen sclerosis caused by autoimmune cross-targeting of spirochetes and HPV16?

Autoimmune cross-targeting hypothesis

The layering of 2 different infections on one target triggering autoimmune disease.  A viral infection marking the inside of the target then a bacterial, or fungal, or mycobacteria infection marking the outside.

Lichen sclerosis trigger by autoimmune cross-targeting of a spirochete and HPV.

spirochetes and lichen sclerosis
http://www.ncbi.nlm.nih.gov/pubmed/2393064
http://www.ncbi.nlm.nih.gov/pubmed/3192771
http://www.ncbi.nlm.nih.gov/pubmed/10792212
I can't tell if this is truly the lyme spirochete or not.

Penile lichen sclerosis and  hpv16
http://www.ncbi.nlm.nih.gov/pubmed/16796627

Penile cancer and lichen sclerosis (the overlap is the virus)
http://www.ncbi.nlm.nih.gov/pubmed/10570372
http://www.ncbi.nlm.nih.gov/pubmed/16515998
http://www.actasdermo.org/en/lichen-sclerosus-squamous-cell-carcinoma/articulo/S1578219012000479/

HPV16 and penile cancer
http://www.webmd.com/cancer/news/20090824/hpv-infections-linked-to-penile-cancer
http://www.ncbi.nlm.nih.gov/pubmed/23474228
http://www.ncbi.nlm.nih.gov/pubmed/18047520 and lichen sclerosis

Talc is a carcinogen.  Use in the penile region during and hpv infection could cause cancer.

Francis Peyton Rous' Co-carcinogenesis hypothesis: that a virus and a carcinogen together cause cancer. (1966 Nobel prize for HPV work)  http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2135410/

What I surmise from his hypothesis:

A virus enters a cell through a receptor, opens up and alters host DNA telomeres. The carcinogen  inhibits the virus' polymerase because viral polymerases have stronger binding affinities than the host's.

Cancer cells can make unlimited copies because of the telomere modifications done by the virus. 

There are DNA polymerases and RNA polymerases. Think of DNA as the cookbook and RNA as recipes...one polymerase copies the entire cookbook, one makes repairs, and one polymerase copies just a recipe.

If the viral polymerases are inhibited by the carcinogen instead of the host's polymerase then the cancer "stem" cell could be created.  The host's polymerases have access to and can make unlimited copies.

Morgellon's filaments and spirochetes
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3257881/
http://www.ncbi.nlm.nih.gov/pubmed/25879673
http://www.ncbi.nlm.nih.gov/pubmed/24715950
http://www.ncbi.nlm.nih.gov/pubmed/16489838
http://www.ncbi.nlm.nih.gov/pubmed/23326202
http://www.ncbi.nlm.nih.gov/pubmed/22253541

specifically morgellon's could be Borrelia garni
http://bmcdermatol.biomedcentral.com/articles/10.1186/s12895-015-0023-0