Schizophrenia as a cross-targeting autoimmune disease

Autoimmune cross-targeting hypothesis: a virus marks the inside of a cell while a larger infection marks the outside and the combination triggers autoimmune disease.  The immune system is instructed to destroy both the inside and the outside of the target.

In the case of schizophrenia the target is the amygdala where t.gondii marks the outside, and then herpes or adenovirus marks the inside.

T.gondii and schizophrenia
http://www.ncbi.nlm.nih.gov/pubmed/17314085
http://www.ncbi.nlm.nih.gov/pubmed/17329231
http://www.ncbi.nlm.nih.gov/pubmed/17404388
http://www.ncbi.nlm.nih.gov/pubmed/17387159
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2526138/

Note that herv seems to appear after a T.gondii infection
http://www.ncbi.nlm.nih.gov/pubmed/17054075?dopt=AbstractPlus 
because herve is a self gene I don't think this triggers autoimmunity

T.gondii appears to mark the outside of the amgydala so which virus could mark the inside (only need one virus to mark the inside and it could be any of the following)

Schizophrenia and CMV
http://www.ncbi.nlm.nih.gov/pubmed/17044725

Schizophrenia and HHV8 or HHV6
http://www.ncbi.nlm.nih.gov/pubmed/24139899
http://www.ncbi.nlm.nih.gov/pubmed/14991372
http://www.ncbi.nlm.nih.gov/pubmed/?term=herpesvirus+8+schizophrenia+2013
http://www.uptodate.com/contents/human-herpesvirus-6-infection-in-hematopoietic-cell-transplant-recipients
http://www.ncbi.nlm.nih.gov/pubmed/24560611

Herpes simplex virus infects the amygdala
http://link.springer.com/article/10.1007%2FBF00868522#page-1

http://www.researchgate.net/publication/7628779_Role_of_the_central_amygdala_in_modulating_the_pituitary-adrenocortical_and_clinical_responses_in_experimental_herpes_simplex_virus-1_encephalitis

Adenoviruses seem to also infect the amygdala
http://www.biomedcentral.com/1471-2202/11/81

adenovirus receptor
https://en.wikipedia.org/wiki/Coxsackie_virus_and_adenovirus_receptor