Acute Disseminated Encephalomyelitis (ADEM): are there multiple types? Autoimmune cross-targeting hypothesis suggests there should be because not all nerves have the same receptors.

Autoimmune cross-targeting hypothesis:  the layering of 2 different infections on one target cell type triggers autoimmune disease.  A viral infection marking the inside of the target then a bacterial, or fungal, or mycobacteria, spirochete infection marking the outside.

Different viral infections can trigger ADEM and it seems logical that since these virus families use different receptors to enter the nerves....there should be different types of ADEM.

Zika and ADEM
http://www.medscape.com/viewarticle/861811

Zika could be using the ACTH receptor

ADEM and autism (measles vaccine in common?)
http://www.huffingtonpost.com/robert-f-kennedy-jr-and-david-kirby/vaccine-court-autism-deba_b_169673.htm

ADEM after vaccination for / or infection with measles, mumps, or rubella
http://www.ncbi.nlm.nih.gov/pubmed/19179725

 Paramyxoviridae (measles family viruses) bind Muscarinic receptors

ADEM has been PROVEN to be triggered by the rabies vaccine

ADEM has also been linked to Enteroviruses which bind Nicotinic acetylcholine receptors

ADEM and enteroviruses
http://www.ncbi.nlm.nih.gov/pubmed/20677593
http://www.ncbi.nlm.nih.gov/pubmed/25576193
http://www.ncbi.nlm.nih.gov/pubmed/17106158
http://www.ncbi.nlm.nih.gov/pubmed/11419405

Multiple sclerosis has associations with herpes zoster which by using the beta estrogen receptor hides in the mitochondria of the nerves.  Thus this form of autoimmune disease could possibly wax and wane with the herpes zoster virus.

Pervious blog posts about MS connect the mycobacteria of psoriasis with the herpes zoster virus as the possible triggers of MS.

It is possible that some cases of ADEM are mycobacterias crossed with other viruses that do not wax and wane. (I had connected high cholesterol and type 2 diabetes to the quorum of mycobacterias : cGMP so this might be the vulnerable group)

The paralysis caused by enteroviruses like polio and D68 are very likely to be autoimmune in nature because not everyone who catches these viruses develop paralysis.  An autoimmune cross-targeting of the spinal region instead of the brain?

Any thoughts?