Transglutaminase2 and Celiac diseae

The culprit infections that could be triggering celiac disease: e.coli and campylobacter could be what is triggering the anti-transglutaminase2 antibodies.  Could what we are seeing be cross-reactivity? The antibodies could actually be generated against the bacteria at their binding sites. The binding sites for the blood group antigens which are expressed not just on blood groups but on the intestinal lining. Is this possible?

Previous blog about celiac disease and infections
http://angelabiggs.blogspot.com/2016/01/updated-analysis-of-infections.html

campylobacter binding to blood group antigens
http://www.jbc.org/content/278/16/14112.full

e.coli and blood group antigens
http://www.ncbi.nlm.nih.gov/pubmed/3127953
http://www.ncbi.nlm.nih.gov/pubmed/2037790

histo-blood group
http://www.ncbi.nlm.nih.gov/pubmed/26191052

transglutaminase 2 and celiac disease
http://www.ncbi.nlm.nih.gov/pubmed/22437759

Coagulation factor XIII and transglutaminase 2 are family isozymes. Do they have shared binding sites?

Does the coagulation factor clot by grabbing the red blood cells' antigens?

Are the antibodies to transglutaminase 2 because it binds blood group antigens?

The infections bind blood group antigens

coagulation factor binds these....where are the blood group antigens here?
http://www.jbc.org/content/289/10/6526.full