Follicular dendritic lymphoma and Co-carcinogenesis

Co-carcinogenesis: a virus and a carcinogen together cause cancer. Taking Francis Peyton Rous’ hypothesis further, the carcinogen is a polymerase inhibitor which binds the viral polymerase with higher affinity. Thus the DNA methylation state has been changed but the virus is no longer viable.  DNA damage does not cause most cancers, the interaction of carcinogens and viruses do.  Further these cancers wear the receptors that the virus that triggered it used to enter and the characteristics of the cancer will be from the receptor's pathways.

follicular lymphoma
http://www.cancerresearchuk.org/about-cancer/follicular-dendritic-cell-fdc-sarcoma/about

Castleman's disease and HHV8
https://en.wikipedia.org/wiki/Castleman%27s_disease

HHV8 is the Kaposi's sarcoma virus
https://en.wikipedia.org/wiki/Kaposi%27s_sarcoma-associated_herpesvirus

vaccine against follicular lymphoma
https://www.news-medical.net/news/2005/11/09/14390.aspx

Tattoo ink as the carcinogen of the lymph nodes
https://www.researchgate.net/publication/230570212_Tattoo_Pigment_Lymphadenopathy_Mimicking_Metastasis_in_Vulvar_Cancer

With the current popularity of Tattoos will this form of cancer increase?

HHV8 and the alpha estrogen receptor
http://d-scholarship.pitt.edu/13517/

alpha estrogen receptor is a marker for follicular dendritic cells
https://www.ncbi.nlm.nih.gov/pubmed/14507640

In order for Cancer to occur the virus must trigger the cell division cycle.  Knudson's Retinoblastoma protein genes are the target of nuclear viral proteins.
http://www.nature.com/onc/journal/v25/n38/full/1209621a.html

Knudson's 2 hit RB cancer gene is the lynch pin to the cell division cycle start
http://angelabiggs.blogspot.com/2017/10/knudsons-heredity-2-hit-cancer-compared.html
(this form does not involve a viral start but most cancers will need a virus to pull this lynch pin)