Lon enzyme has involvement in virulence, morphology, and is stimulated by casein...gluten too?

This hypothesis was disproven. T.gondii and strep are not dimorphic. See newer gluten posts.

LON is the enzyme I am attempting to associate with gluten and casein for the dimorphic switching of infections...causing our immune systems to develop sensitivity to them.

NEWER post: http://angelabiggs.blogspot.com/2014/03/lon1-and-dimorphic-switching-caused-by.html

lon enzyme
http://www.ncbi.nlm.nih.gov/pubmed/15501647 

E.coli Lon and morphology (changes between rod and filament forms)
http://www.ncbi.nlm.nih.gov/pubmed/23391222

Campylobactor (lon protein contributes to virulence)
http://www.ncbi.nlm.nih.gov/pubmed/17933920

Mycobacteria Lon
 casein sensitive
http://www.ncbi.nlm.nih.gov/pubmed/11045626  
http://www.ncbi.nlm.nih.gov/pubmed/9698372

Mycobacteria are dimorphic (changes morphology like e.coli does with LON into a cording form)
http://mic.sgmjournals.org/content/26/1/97

T.gondii and proteases and virulence ? (it might not be t.gondii but mycobacteria with this form of  schizophrenia )
http://www.ncbi.nlm.nih.gov/pubmed/22202120

not sure how this fits:  HU is involved with t.gondii virulence ( mutated in e.coli HU causes filamentation)
http://ec.asm.org/content/11/7/905.full
lon protease degrades HU...so would this explain gluten/casein and t.gondii?
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC298136/

or there is more than one type of schizophrenia which is the most logical conclusion given that Tuberculosis has a risk of schizophrenia
http://www.biomedcentral.com/1471-2334/13/351