Is Celiac disease triggered by autoimmune cross-targeting?

Autoimmune cross-targeting hypothesis: a virus marks the inside of a cell while an infection marks the outside and the combination triggers autoimmune disease.

In celiac disease the infected cells are the intestinal epithelial lining.  The infections do not cause the disease they trigger the autoimmunity.

The bacterial signature in celiac suggests e.coli is there
http://www.biomedcentral.com/1471-2180/10/175

There exists a history of bladder infections in celiac patients (most bladder infections are e.coli)
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1511510/

The virus in celiac disease is astrovirus or hepatitis C
http://www.ncbi.nlm.nih.gov/pubmed/17549632
http://www.ncbi.nlm.nih.gov/pubmed/15302945 (expressed in intestinal epithelial)
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2937664/

Infections that cross have the ability to cross the intestinal or blood brain barriers trigger gluten sensitivity.  Gluten is a macromolecule which is huge and it cannot cross the intestine unless the tight junction protein claudin between cells have been destroyed.

http://angelabiggs.blogspot.com/2015/03/infections-that-break-tight-junction.html

E.coli is one of these tight junctions breakers
http://www.ncbi.nlm.nih.gov/pubmed/16127426

Claudin 5 and the tight junctions between endothelial cells which control the vascular permeability at the intestine
http://jcb.rupress.org/content/147/1/185.full.pdf

Gluten if it leaks across amplifies immune reactions.  For celiac disease the autoimmune disease is occurring right on top of the barrier leak which  can intensify the immune response....or at least that is what I am suggesting.

gluten stimulates cytokines
http://www.ncbi.nlm.nih.gov/pubmed/9721152

Gluten sensitivity exists at the blood brain barrier not just the intestine (again gluten/gliadin is too big as a macromolecule which means another infection must be creating the hole)
http://www.ncbi.nlm.nih.gov/pubmed/22512541