If endometriosis is connected to ulcerative colitis? How and why? (updating 2018)
Link of endometriosis and ulcerative colitis and crohn's?
https://www.ncbi.nlm.nih.gov/pubmed/26332310
https://www.medicinenet.com/script/main/art.asp?articlekey=152823
https://www.ncbi.nlm.nih.gov/pubmed/22184069
C.sordellii releases an acid like h.pylori, does this cause the ulcers of ulcerative colitis?
C. diff not C.sordellii? isolated from colitis
https://www.ncbi.nlm.nih.gov/pubmed/700321
https://www.ncbi.nlm.nih.gov/pubmed/625309
http://pmj.bmj.com/content/postgradmedj/63/745/955.full.pdf
http://www.gastrojournal.org/article/0016-5085(79)90345-7/abstract
specialized uterine NK cells
https://www.ncbi.nlm.nih.gov/pubmed/17100884/
estrogen controls the migration of these uNK cells and it's secretion of CCL2
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4498222/
Estrogen and the immune system: estrogen must drop for fertilization to occur
http://www.encognitive.com/node/3776
If Estrogen is kept high is that because a bacteria exists in the area and the immune system wants to keep uNK active?
il-22 and endometriosis
https://www.ncbi.nlm.nih.gov/pubmed/24133578
il-22 is the vacuole popping cytokine released by th17
il-22 and C.diff
https://www.ncbi.nlm.nih.gov/pubmed/25367575
c.diff in vacuoles
http://iai.asm.org/content/77/12/5478.full
endometriosis cytokines: CCL8 (which is il-8) ,CCL2(mcp-1), CCL5 (RANTES)
https://www.ncbi.nlm.nih.gov/pubmed/24287816
CCL2 induces angiogenesis (blood vessel growth)
https://www.ncbi.nlm.nih.gov/pubmed/16888027
https://www.ncbi.nlm.nih.gov/pubmed/22265030
il-8 is the " bacteria released a TOXIN" cytokine? calling the neutrophils?
il-8 is known at the neutrophil chemotactic factor
neutrophils are critical for the early response against these types of pathogens
immune responses during toxic shock syndrome/ toxins
http://iai.asm.org/content/81/5/1751.full
http://iai.asm.org/content/77/3/1182.full
il-8 and h.pylori
http://iai.asm.org/content/73/3/1523.abstract
c.diff toxins and il-8
http://www.ncbi.nlm.nih.gov/pubmed/9435559
shiga toxin from e.coli and il-8
http://www.ncbi.nlm.nih.gov/pubmed/10531258
C.sordelii does release a toxin
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC415707/
CCL5 is a chemoattractant calling Eosinophils (histamine release) Basophils and T helper cells
https://www.sciencedirect.com/topics/neuroscience/ccl5
CCL5 aka RANTES causes the inflammation
TLR4 and clostridium
http://journals.plos.org/plospathogens/article?id=10.1371/journal.ppat.1002076
TLR4 and endometriosis
https://www.ncbi.nlm.nih.gov/pubmed/18596029
TLR4 binds lipopolysaccharides of bacteria : cytokines it can release IL-6, IL-8, CXCL1, CXCL2, CXCL3, and CCL2
http://www.jbc.org/content/289/4/2457.full
c.diff cytokines: IL-1α, IL-1β, IL-6, IL-8 and tumour necrosis factor-α
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4040718/
Ulcerative colitis and chamomile/daisy allergy: the natural remedy
endometriosis and ragweed allergy
https://www.ncbi.nlm.nih.gov/pubmed/22369407
Link of endometriosis and ulcerative colitis and crohn's?
https://www.ncbi.nlm.nih.gov/pubmed/26332310
https://www.medicinenet.com/script/main/art.asp?articlekey=152823
https://www.ncbi.nlm.nih.gov/pubmed/22184069
C.sordellii releases an acid like h.pylori, does this cause the ulcers of ulcerative colitis?
C. diff not C.sordellii? isolated from colitis
https://www.ncbi.nlm.nih.gov/pubmed/700321
https://www.ncbi.nlm.nih.gov/pubmed/625309
http://pmj.bmj.com/content/postgradmedj/63/745/955.full.pdf
http://www.gastrojournal.org/article/0016-5085(79)90345-7/abstract
specialized uterine NK cells
https://www.ncbi.nlm.nih.gov/pubmed/17100884/
estrogen controls the migration of these uNK cells and it's secretion of CCL2
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4498222/
Estrogen and the immune system: estrogen must drop for fertilization to occur
http://www.encognitive.com/node/3776
If Estrogen is kept high is that because a bacteria exists in the area and the immune system wants to keep uNK active?
il-22 and endometriosis
https://www.ncbi.nlm.nih.gov/pubmed/24133578
il-22 is the vacuole popping cytokine released by th17
il-22 and C.diff
https://www.ncbi.nlm.nih.gov/pubmed/25367575
c.diff in vacuoles
http://iai.asm.org/content/77/12/5478.full
endometriosis cytokines: CCL8 (which is il-8) ,CCL2(mcp-1), CCL5 (RANTES)
https://www.ncbi.nlm.nih.gov/pubmed/24287816
CCL2 induces angiogenesis (blood vessel growth)
https://www.ncbi.nlm.nih.gov/pubmed/16888027
https://www.ncbi.nlm.nih.gov/pubmed/22265030
il-8 is the " bacteria released a TOXIN" cytokine? calling the neutrophils?
il-8 is known at the neutrophil chemotactic factor
neutrophils are critical for the early response against these types of pathogens
immune responses during toxic shock syndrome/ toxins
http://iai.asm.org/content/81/5/1751.full
http://iai.asm.org/content/77/3/1182.full
il-8 and h.pylori
http://iai.asm.org/content/73/3/1523.abstract
c.diff toxins and il-8
http://www.ncbi.nlm.nih.gov/pubmed/9435559
shiga toxin from e.coli and il-8
http://www.ncbi.nlm.nih.gov/pubmed/10531258
C.sordelii does release a toxin
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC415707/
CCL5 is a chemoattractant calling Eosinophils (histamine release) Basophils and T helper cells
https://www.sciencedirect.com/topics/neuroscience/ccl5
CCL5 aka RANTES causes the inflammation
TLR4 and clostridium
http://journals.plos.org/plospathogens/article?id=10.1371/journal.ppat.1002076
TLR4 and endometriosis
https://www.ncbi.nlm.nih.gov/pubmed/18596029
TLR4 binds lipopolysaccharides of bacteria : cytokines it can release IL-6, IL-8, CXCL1, CXCL2, CXCL3, and CCL2
http://www.jbc.org/content/289/4/2457.full
c.diff cytokines: IL-1α, IL-1β, IL-6, IL-8 and tumour necrosis factor-α
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4040718/
Ulcerative colitis and chamomile/daisy allergy: the natural remedy
endometriosis and ragweed allergy
https://www.ncbi.nlm.nih.gov/pubmed/22369407
No comments:
Post a Comment