How do Herpes viruses enter into cells do they use the estrogen receptors or the Herpes-entry-mediator?

Viruses are extremely savvy.  Some viruses have evolved to bind the TAM hands of macrophages to  block the destruction of virally infected cells. (Macrophages are the immune system's vacuum)

The "herpesvirus entry mediator" is a TNF receptor. This is not the primary mode of entry of the virus rather an attempt to block the immune response.

TNF alpha is released by macrophages when they are themselves infected. (and unable to function) By blocking the receptors for TNF the viruses are trying to "block" the knowledge that macrophages are infected and stop the activation of neutrophils etc.

Neutrophils are the strongest defense of the intestine
https://www.ncbi.nlm.nih.gov/pubmed/22491176

Neutrophils do not have transcription or translation abilities and can therefore deal with viral infections better than macrophages.

Herpesvirus entry mediator
https://link.springer.com/article/10.1245%2Fs10434-017-5924-1

this TNF receptor has high levels in the mucosal membranes
https://www.ncbi.nlm.nih.gov/pubmed/24851095

I still contest that the main mode of Herpes viral entry on regular cells is through estrogen receptors.

http://angelabiggs.blogspot.com/2016/08/attempting-to-match-up-herpes-viruses.html

Alpha-herpes viruses: Herpes simplex 1, herpes simplex 2,  herpes zoster : Estrogen-beta receptors (nerves and uterine tissue)

Beta-herpes viruses: CMV, HHV6, HHV7 :  Estrogen-related receptors (CMV binding confirmed)

Gamma-herpes viruses: EBV, HHV8 : Estrogen-alpha receptors (lymphocytes, breast involved

Estrogen-related receptors and estrogen-alpha receptors cycle to the nucleus which is why these herpes viruses can cause cancer while the Beta-estrogen receptors cycle to the mitochondria where alpha herpes viruses can hide for years.