MCR4, Hep C, glucose metabolism, and HCC (heptocellular carcinoma): pattern reveals a distinctive Type2 diabetes

liver cells stop making glucose as they become cancerous
https://www.sciencedaily.com/releases/2012/07/120730141635.htm

MCR4 and glucose metabolism...so so through insulin?
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3033043/

Hepatitis C, insulin resistance, and HCC
https://www.ncbi.nlm.nih.gov/pubmed/20458764

Hepatitis C and insulin resistance
https://www.sciencedaily.com/releases/2010/03/100309102519.htm

melanocortin hormone and insulin resistance
https://link.springer.com/article/10.2165/00024677-200605010-00002

the glucose transporter suppresses the melanocortin receptor
http://www.endocrine-abstracts.org/ea/0041/ea0041EP809.htm

melanocortin increases GLUT expression? and alters glucose metabolism?

melanocortin antagonist increases insulin sensitivity
http://www.sciencedirect.com/science/article/pii/S0196978104002050
http://www.sciencedirect.com/science/article/pii/S0014579307001652

over expression of agouti caused increased growth not obesity
https://www.ncbi.nlm.nih.gov/pubmed/27156808

HCC and GLUT-2 (glucose transporter)
https://www.ncbi.nlm.nih.gov/pubmed/18949368

HCC and GLUT-1
https://www.ncbi.nlm.nih.gov/pubmed/19874261
https://www.ncbi.nlm.nih.gov/pubmed/19286567

(so there must be two types of type 2 diabetes: hepatitis C type and the mycobacteria type)

hep C and type 2 diabetes
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2669937/

The mycobacteria type overlaps psoriasis, chron's, parkinson's and has fatty liver disease with high cholesterol.  The issue with mycobacteria is that the quorum is cGMP which messes with our cells glucose transporter.

In the case of hep C the triggering of the melanocortin 4 receptor pulls the glucose transporters from the membrane and changes the glucose metabolism through gene changes. We should be able to sort these 2 types of diabetes out from each other.

Hep C causes the liver to scar (cirrhosis) over time.  The liver cells are not just dysfunctional they are dying.

Damage to other organs infected by hep C  has started to emerge.

Pulmonary fibrosis (scarring lung damage)
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3206667/

Scarring of the colon could be what causes diverticulitis, the scars cause the colon to fold, explaining the "left colon cancer" connection
https://www.ncbi.nlm.nih.gov/pubmed/8555344

The non-Hodgkin's lymphoma called nodular sclerosis, which is the most common kind, is filled with fibrous scars.
https://en.wikipedia.org/wiki/Nodular_sclerosis

hep c and non-Hodgkin's lymphoma
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4802504/

overlap of B cell and nodular lymphoma
https://bmccancer.biomedcentral.com/articles/10.1186/1471-2407-14-332

Liver, colon, and lung cancers have been linked to hep C....along with a huge list of other cancers.

other overlapping disorders linked to hep C can help us separate the diabetes:

http://emedicine.medscape.com/article/1134161-clinical?pa=bNC91gt1rcVQirt7Fvkd%2FYuDdd7pFGHrK2nx0uRZFmz1FjlQja9m4jLs8Nzam%2B%2BuMvFliwSOoozYUdBPs1l8Qd1zi39gDfdraEJUUFno0Ig%3D

Lichen planus is the skin manifestation of hep C

And because Hep C also uses MCR1 Thrombocytopenia (red blood cells) can appear

The point is that the type 2 diabetes associated with Hep C will have completely different issues from the mycobacteria form.