il-17, Neutrophils, and fungal infections

Th17 cells are involved with the second popping of infections hiding inside host cells.

The il-17 cytokines associated with Th17 are also involved with killing extremely large infections where il-17 up regulates neutrophils and CTLs

il-17a il-17f and neutrophils

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2759196/

neutrophils kill fungal infections

https://www.ncbi.nlm.nih.gov/pubmed/26718340

https://www.ncbi.nlm.nih.gov/pubmed/27558342

https://www.ncbi.nlm.nih.gov/pubmed/25794823

C-type lectin receptors on macrophages, dendritic cells, and neutrophils sense fungal infections. Dectin-1 detects B13glucan on candida, aspergillus, and saccharmyces. Dectin-2 binds high mannose structures on cryptococcus and trichophyton. 

Dectin-1 

https://www.ncbi.nlm.nih.gov/pubmed/21803640

neutrophils produce il-17a in a dectin-1 

https://www.ncbi.nlm.nih.gov/pubmed/21807912

il-17a induces CTL cells which have been shown to kill not just host cells infected with viruses but fungal infections

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2896662/

il-17, TH17, and candida
https://www.sciencedirect.com/science/article/pii/S1931312809000936

fungal infections trigger the release of TGF-b1

https://www.ncbi.nlm.nih.gov/pubmed/11447193

My current cytokine hypothesis for TH17 cells