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Hypothesis is that there exists 2 stages of autoimmunity: first an infection that builds up antibodies then a virus which causes the immune system to cross-target on self.
Separating the 2 stages of autoimmune disease.
The allo-antibodies avidity compared to the auto-antibodies that are present in celiac disease. why would e.coli be so strong? is it because e.coli is the first infection?
http://www.ncbi.nlm.nih.gov/pubmed/24666357
Parkin connecting parkinson's and TB
https://www.ucsf.edu/news/2013/09/108621/tb-and-parkinson%E2%80%99s-disease-linked-unique-protein
( i believe parkinson's is an autoimmune disease and new research supports it: http://www.sciencedaily.com/releases/2014/04/140417151227.htm )
RA and mycoplasmas
http://www.ncbi.nlm.nih.gov/pubmed/5165178
http://www.ncbi.nlm.nih.gov/pubmed/4948122
sjogren's and candida
http://www.ncbi.nlm.nih.gov/pubmed/12973284
sj and viral trigger
http://www.ncbi.nlm.nih.gov/pubmed/24774584
Hypothesis is that there exists 2 stages of autoimmunity: first an infection that builds up antibodies then a virus which causes the immune system to cross-target on self.
Separating the 2 stages of autoimmune disease.
The allo-antibodies avidity compared to the auto-antibodies that are present in celiac disease. why would e.coli be so strong? is it because e.coli is the first infection?
http://www.ncbi.nlm.nih.gov/pubmed/24666357
Parkin connecting parkinson's and TB
https://www.ucsf.edu/news/2013/09/108621/tb-and-parkinson%E2%80%99s-disease-linked-unique-protein
( i believe parkinson's is an autoimmune disease and new research supports it: http://www.sciencedaily.com/releases/2014/04/140417151227.htm )
RA and mycoplasmas
http://www.ncbi.nlm.nih.gov/pubmed/5165178
http://www.ncbi.nlm.nih.gov/pubmed/4948122
sjogren's and candida
http://www.ncbi.nlm.nih.gov/pubmed/12973284
sj and viral trigger
http://www.ncbi.nlm.nih.gov/pubmed/24774584
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