Combining the T gammadelta hypothesis with the autoimmune cross-targeting hypothesis.

The T gamma delta hypothesis was that Tgd function like B cells to produce gamma delta antibodies against lipid antigens which get confused with the IgG1 of B cells.    What is important is the idea that these are completely different immune system "couples". Instead of T and B it is NKT and T gamma delta.

Surmised cytokines

In order to create IgG4 the B cell must switch T cell partners and see a different cytokine. There is confusion and mixing. Tfh making il-4 for the outer antigens of E.coli while at the same time Tc would be making il-10? 

Possible? 

Can we show that these are Gamma delta antibodies and not IgG1?

Just for fun : IgG gamma delta mycobacteria (cytosol lipid infection)
                      IgG gamma delta Bacteria ( visible lipid  infection )
                      IgG gamma delta virus (lipid coat)
I just made this up....right now they are all lumped together as IgG1

3 different gamma delta elements in the T gamma delta receptor
https://www.ncbi.nlm.nih.gov/pubmed/7511639
                   

Gram positive Bacteria lipid A held by cd1.c  and cd1.b (monocytes and macrophages)
https://www.ncbi.nlm.nih.gov/pubmed/19948070

cd1.b on macrophages (cdl.b induced by il-4)
https://www.ncbi.nlm.nih.gov/pubmed/9175583

So cdl.c and cdl.b  both end up producing the IgG gamma delta B ?

HIV and cd1.a and langerhans
https://www.ncbi.nlm.nih.gov/pubmed/17442711