How does gluten fit into the autoimmune cross-targeting hypothesis?

New hypothesis: Two separate zones for inner and outer antigens in lymph organs.

ILF the inducible lymph follicle deals with the inner antigens while the peyer patch deals with the outer antigens of the mucosal membranes.

ILF  https://www.ncbi.nlm.nih.gov/pubmed/12759424

E.coli infections would involve the outer pathways. At the mucosal's peyer patch they would see high growth hormone GH thus produce IgA.

According to the Cross-targeting hypothesis when a second inner infection appears and calls the B cell to the ILF zone and away from the peyer patch....the B cell becomes a regulatory autoimmune B cell which secretes IgG4 antibodies that stop the outer pathways.

GH-IGF axis and gluten in celiac
https://www.karger.com/article/pdf/185499

Gluten was seen as the infection not the parasite because the parasite moved inside of cells?

UPEC :the e.coli that moves inside cells
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5244466/

If patients remove gluten and outside pathway is stopped? Is that because the infection is on the inside of cells but gluten was seen as the infection?

Note that the anti-gluten antibody is not IgG4 but IgG1.

gluten and e.coli in mice
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4630176/

e.coli and celiac
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6287676/

? protein from e.coli binds gliadin?
https://www.ncbi.nlm.nih.gov/pubmed/16944918

Do these infection hold gluten on the outside like a cloak?

Anti-gluten response after t.gondii infection in mice?
https://www.ncbi.nlm.nih.gov/pubmed/23209841

anti-gluten and t.gondii
https://www.ncbi.nlm.nih.gov/pubmed/22446142