1. Allergy hypothesis: Allergies reveal the infections we have.
Severe allergies and infections can be matched up: peanut and Staph, bee
and T.gondii, daisy and Clostridium, poison ivy and spirochetes. If an
infection can be killed by a compound or inhibited by the compound our immune
system sees the interaction and we associate all the compounds present with the
infection. If an infection makes a pigment from a compound we will react
to that too. For example aflatoxin with peanuts could cause staph to counter
respond. Our immune system would identify everything there at the scene
of the crime as part of the infection.
2. Quorum hypothesis: Infections talk using quorums and these
quorums interfere with our body's pathways. ( for example mycobacteria's cGMP
causes such things as type 2 diabetes, high cholesterol, and synuclein
bodies)
3.. Autoimmune cross-targeting hypothesis: The
Cross-targeting of simultaneous infections on one tissue, one infection inside
and one infections outside, triggers autoimmunity. Antibodies or
chemicals/drugs can replace one of the infections. By antibodies I mean
even those triggered by vaccines. For
example a flu virus inside of the pancreas and e.coli latching on to the
outside could trigger type one diabetes.
4. Co-carcinogenesis: a virus and a carcinogen together cause
cancer. Taking Francis Peyton Rous’ hypothesis further, the carcinogen is a polymerase
inhibitor which binds the viral polymerase with higher affinity. Thus the DNA
methylation state has been changed but the virus is no longer viable. DNA
damage does not cause most cancers, the interaction of carcinogens and viruses
do. Further these cancers wear the
receptors that the virus that triggered it used to enter and the
characteristics of the cancer will be from the receptor's pathways.
5. Barrier crossing infections are ones that can cross the intestine
or the blood brain barrier causing gluten sensitivity with the hole they leave
behind. This explains the gluten sensitivity of Schizophernia from T.gondii,
Celiac disease from e.coli, Tourettes from staph, and crohn’s from mycobacteria.
6. Viral families use receptor families. Influenzas use dopamine
receptors. HPVs use Cannabinoid receptors. Herpes viruses use estrogen and
estrogen-like receptors. Flaviviruses use melanocortin receptors.
Polyomaviruses use serotonin receptors.
7. Aflatoxin like compounds cause tau bodies. ALS and picks
disease are caused by different infections but both have tau. Both
infections secrete an aflatoxin like compound. Vitamin D is protective
against it because aflatoxin uses the vit D receptor.
8. Pituitary tumors are caused by an infection releasing butyrolactones
which interfere with normal GABA signals and cause the immune system to confuse
nerves with the infection. (or someone taking too many GHB sleeping pills)
9. The receptors used by a virus when triggering
autoimmunity cause pathways to be activated which makes the various types of
one autoimmune disease. There are 3 types of schizophrenia which match up
with the 3 receptors use by the 3 herpes viruses. (sort of a continuation of 6)
The receptor pathway triggered by a virus involved in an
autoimmune disease can cause symptoms of the disease that will be distinct. Example:
The 3 types of schizophrenia has all 3 herpes virus families and each
receptor activated can be matched to symptoms. Estrogen-related receptor
and disorganized symptoms for example. 3 receptors used equals 3 types of
schizophrenia
10. Alzheimer's disease is caused by damage to the mitochondria:
alpha-herpes family, diacetyl, inherited and linked to down syndrome, or
radiation damaged mitochondria.
11. Postural orthostatic tachycardia could be caused by reoviruses
and the adrenergic receptors they bind.
12. The HLAs are mailboxes for T cells from different areas
of the cell. HLA-A is the nuclear mailbox. HLA-B is the mitochondria's mailbox.
HLA-C is the endoplasmic reticulum's mailbox. HLA-DR is the cytosol's mailbox
for RNA viruses. HLA-DQ is the cytosol's mailbox for non-encapsulated viruses
there. HLA-DP is the plasama membrane mailbox.
13. Carcinogens use certain receptors to get into cells which
matches them up with specific cancers.
14. TLRs, toll like receptors, are the innane immune system of
nets catching conserved molecules in a non specific way but identifying the
region as well as general type of infection. Like HLAs they exist in the
different areas of the cell. When the internal viral seeking TLRs are
activated the IFN matches the region they are in thus the appropriate HLA is
produced helping to find the exact culprit.
15. TLRs send IFNs which tells infected cells to express the
corresponding HLAs and macrophages to wear the corresponding TAMs (hands to
grab infected cells) TAMS: tyro T , axl A , mer M
Cytosol TLR3
IFNbeta HLA-D
Tyro3
Nucleus/ Mito TLR7/9 IFNalpha
HLA-A HLA-B Mer
Endoplasmic R TLR 8 IFNgamma HLA-C
Axl
(because an ER infected means little gets to the surface of
a cell Natural killer cells step in here and secrete IFNgamma)
16. Nuclear viruses awaken embryonic Hervs. They do so by
methylating or demethylating DNA. If the virus family demethylates the
cancer will be aggressive while the methylating viruses are slow growing.
(Cancer occurs when a carcinogen inhibits the viral polymerase..so the
virus opens the DNA up but can't use it)
