Previously hypothesis of how the lymph is organized in this sketich, so how does nk fit in?
il-18 and neutrophil activation
https://www.ncbi.nlm.nih.gov/pubmed/11509635
il-18 and nk activation
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5154249/
il-18 listeria
https://www.gastrojournal.org/article/S0016-5085(09)63232-7/abstract
listeria and golgi/ER
http://www.jimmunol.org/content/jimmunol/168/7/3428.full.pdf
il-15 and nk
https://www.pnas.org/content/101/47/16606
il-15 receptor and mantle B cells
http://www.jimmunol.org/content/171/2/569
RSV and il-15
https://www.ncbi.nlm.nih.gov/pubmed/17158609
il-15 tells the nk there is a viral infection of the ER and the golgi
il-18 tells the nk there is a bacterial infection of the ER and the golgi
il-15 receptors are on the mantle B cells
il-18 receptors are on the GC B cells
il-18 secreted by dendritic cells and macrophages
https://www.frontiersin.org/articles/10.3389/fimmu.2013.00289/full
il-15 secreted by monocytes and macrophages
mycoplasmas and RA
https://academic.oup.com/rheumatology/article/44/7/912/1788375
mycoplasmas and ER
https://www.ncbi.nlm.nih.gov/pubmed/4844719
ANA antibodies
https://www.rheumatology.org/I-Am-A/Patient-Caregiver/Diseases-Conditions/Antinuclear-Antibodies-ANA
listeria associated arthritis
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1153789/
Is autoantibody IgG4 created when the B cell saw il-18 in the GC area first but then moved to the mantle when exposed to a virus?
Note that the receptor on the B cell matches up with bacterial vs. viral.
il-15 activated NK can help against HIV
https://jvi.asm.org/content/92/12/e00235-18
